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AVOIDANCE or PRESSURE 211
of the white corpuscles through their walls into the interstices
of the pulp tissue.
4. Suppuration.—The corpuscles and the surrounding
pulp tissue are broken down and transformed into pus,
through the action of germs, which have entered by way of the
blood stream.
5. Moist Gangrene.— (Putrefaction.) The blood current
becomes stagnant and finally ceases, on account of the pres-
sure in the vessels passing through the minute apical foramen.
Death of the tissues occurs from lack of nourishment, putre-
factive decomposition and destruction occurring through the
action of germs, with the formation of malodorous substances.
6. Dry Gangrene.—Germs do not enter, death of the tis-
sues occurring without suppuration or putrefaction, from
strangulation and lack of nourishment.
7. Alveolar Abscess.—The putrescent material infects
the tissues of the apical space, with resultant pus formation,
breaking down and destruction of these parts.
The above conditions may be classified as follows, viz.:
I. Vital Pulps.
(A) Pulps capable of being restored to the normal (Arterial
Hyperaemia)
(Venous Hyperemia)
(B) Pulps incapable of being re-
(Inflammation)
stored to the normal.
(Suppuration)
2. De-Vital Pulps. (Non-vital.)
(A) Pulps uninfected. (Dry Gangrene.)
(B) Pulps in a state of putrefaction and infection. (Moist
Gangrene.)
(C) Pulps with apical complications. (Alveolar Abscess.)
Avoidance of Pressure.—In cases of exposure (a minute
opening through the wall of the pulp-chamber) , the vital pulp
rebels against the application at the point of exposure of the
AVOIDANCE or PRESSURE 211
of the white corpuscles through their walls into the interstices
of the pulp tissue.
4. Suppuration.—The corpuscles and the surrounding
pulp tissue are broken down and transformed into pus,
through the action of germs, which have entered by way of the
blood stream.
5. Moist Gangrene.— (Putrefaction.) The blood current
becomes stagnant and finally ceases, on account of the pres-
sure in the vessels passing through the minute apical foramen.
Death of the tissues occurs from lack of nourishment, putre-
factive decomposition and destruction occurring through the
action of germs, with the formation of malodorous substances.
6. Dry Gangrene.—Germs do not enter, death of the tis-
sues occurring without suppuration or putrefaction, from
strangulation and lack of nourishment.
7. Alveolar Abscess.—The putrescent material infects
the tissues of the apical space, with resultant pus formation,
breaking down and destruction of these parts.
The above conditions may be classified as follows, viz.:
I. Vital Pulps.
(A) Pulps capable of being restored to the normal (Arterial
Hyperaemia)
(Venous Hyperemia)
(B) Pulps incapable of being re-
(Inflammation)
stored to the normal.
(Suppuration)
2. De-Vital Pulps. (Non-vital.)
(A) Pulps uninfected. (Dry Gangrene.)
(B) Pulps in a state of putrefaction and infection. (Moist
Gangrene.)
(C) Pulps with apical complications. (Alveolar Abscess.)
Avoidance of Pressure.—In cases of exposure (a minute
opening through the wall of the pulp-chamber) , the vital pulp
rebels against the application at the point of exposure of the
